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This study was designed to characterize the multi-level nutritional responses to dietary DHM supplementation, with emphasis on hepatic redox–inflammatory status, jejunal barrier-related phenotypes, cecal microbiota remodeling, hepatic metabolomic alterations, and homocysteine (Hcy) metabolism-related markers in mice

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P53 regulates TIGAR, which is associated with decreased NAD+/NADH redox couple [93, 94]